Monday, February 11, 2013

Question

1) Can Ocular NSAIDS (e.g ketorolac eye drops) cause AKI?

2) Can Topical NSAIDS (e.g diclofenac skin cream) cause AKI ?  

3 comments:

  1. There is one case reports which i could find of patient with ARF on HD coming off dialysis on stopping topical NSAIDs and one case report of biopsy showing FSGS with possible interstitial nephritis in a patient using topical NSAIDs with Cr and Proteinuria improving off topical NSAIDs.
    I guess theoretically ocular ones can also cause it but, I didn't find anything reported.

    ReplyDelete
  2. Patients with ARF on HD come off dialysis all the time - my guess is that the recovery of renal function was unrelated to discontinuation of the topical NSAID.

    I found one large study of adverse events with topical NSAIDs for arthritis in patients with multiple comorbidities - ARF was not even mentioned/reported as an adverse outcome.

    In the absence of good data, my hunch is that systemic absorption is pretty low and unlikely to cause renal dysfuction - at least, not via the typical mechanism afferent arteriolar constriction.

    ReplyDelete
  3. Following application of ketorolac tromethamine 0.5% ophthalmic drops 3 times/day, the mean plasma concentrations of ketorolac are ~4% to 8% of the minimum concentration observed following oral doses of 10 mg 4 times/day.........So may not be clinically significant to cause renal effects.


    Diclofenac topical gel has 6% to 10% systemic absorption. The prescribing information did not specifically mention about AKI but mention "No information is available from controlled
    clinical studies regarding the use of Voltaren® Gel in patients with advanced renal disease. Therefore, treatment with Voltaren® Gel is not recommended in patients with advanced renal disease. If Voltaren® Gel therapy is initiated, close monitoring of the patient’s renal function is
    advisable."

    ReplyDelete